Category Archives: Cardiac Remodeling

Cardiac remodeling is generally accepted as a determinant of the clinical course of heart failure (HF). Defined as genome expression resulting in molecular, cellular and interstitial changes and manifested clinically as changes in size, shape and function of the heart resulting from cardiac load or injury, cardiac remodeling is influenced by hemodynamic load, neurohormonal activation and other factors still under investigation Continue reading →

Ventricular hypertrophy (VH) is thickening of the walls of a ventricle (lower chamber) of the heart.[1][2] Although left ventricular hypertrophy (LVH) is more common, right ventricular hypertrophy (RVH), as well as concurrent hypertrophy of both ventricles can also occur. Ventricular hypertrophy can result from a variety of conditions, both adaptive and maladaptive. Continue reading →

In cardiology, ventricular remodeling (or cardiac remodeling)[1] refers to changes in the size, shape, structure, and function of the heart. This can happen as a result of exercise (physiological remodeling) or after injury to the heart muscle (pathological remodeling).[2] The injury is typically due to acute myocardial infarction (usually transmural or ST segment elevation infarction), but may be from a number of causes that result in increased pressure or volume, causing pressure overload or volume overload (forms of strain) on the heart. Continue reading →

Cardiac remodeling is a term that refers to changes in the hearts size and shape that occur in response to cardiac disease or cardiac damage. When doctors talk about remodeling, they are usually talking about the left ventricle, though occasionally this term is applied to other cardiac chambers. When you remodel your house, that is often considered a good thing. Continue reading →

Cardiac remodeling is a term that refers to changes in the hearts size and shape that occur in response to cardiac disease or cardiac damage. When doctors talk about remodeling, they are usually talking about the left ventricle, though occasionally this term is applied to other cardiac chambers. When you remodel your house, that is often considered a good thing. The opposite is true with remodeling of the heart. In general, the greater the degree of ventricular remodeling, the worse the patients outcome is likely to be. When the left ventricle is damaged -for instance, by amyocardial infarction(heart attack) or by cardiomyopathy-changes often occur in the size and shape of the ventricle. The ventricle tends to become enlarged, its general shape becomes more globular and less elliptical, and the muscular wall of the ventricle often becomes thinner. This remodeling occurs due to mechanical stress on the heart muscle produced by the underlying disease process. In the early stages of a heart attack, some degree of remodeling can help the ventricle compensate for the damage that has occurred. But if this initial remodeling process continues, and the changes in the size and shape of the ventricle become more exaggerated, cardiac function … Continue reading →

An implanted cardiac resynchronization device is a medical device used in cardiac resynchronization therapy (CRT). It resynchronizes the contractions of the hearts ventricles by sending tiny electrical impulses to the heart muscle, which can help the heart pump blood throughout the body more efficiently. CRT defibrillators (CRT-D) also incorporate the additional function of an implantable cardioverter-defibrillator, to quickly terminate an abnormally fast, life-threatening heart rhythm. CRT and CRT-D have become increasingly important therapeutic options for patients with moderate and severe heart failure.[1] Cardiac resynchronization therapy (CRT) is an effective therapy in patients with heart failure and dyssynchrony identified as a prolonged QRS duration.[2] Benefits include improved exercise tolerance, reduced remodeling (reduction in LV chamber size and measures of mitral regurgitation), reduced mortality, and reduced need for hospitalization in patients in sinus rhythm. Several research papers[3][4] have proposed software platforms for planning and guiding the implantation of CRT devices. This research proposes using pre-operative images to characterize tissue and left ventricle activation to identify potential target regions for deploying the CRT leads. View original post here: Cardiac resynchronization therapy - Wikipedia … Continue reading →

Kuyumcu G, Salazar GM, Prabhakar AM, Ganguli S. Minimally invasive treatments for perforator vein insufficiency. Cardiovasc Diagn Ther. 2016 Dec;6(6):593-598. doi: 10.21037/cdt.2016.11.12. Review. PubMed PMID: 28123979; PubMed Central PMCID: PMC5220201. Baliyan V, Tajmir S, Hedgire SS, Ganguli S, Prabhakar AM. Lower extremity venous reflux. Cardiovasc Diagn Ther. 2016 Dec;6(6):533-543. doi: 10.21037/cdt.2016.11.14. Review. PubMed PMID: 28123974; PubMed Central PMCID: PMC5220199. Zucker EJ, Ganguli S, Ghoshhajra BB, Gupta R, Prabhakar AM. Imaging of venous compression syndromes. Cardiovasc Diagn Ther. 2016 Dec;6(6):519-532. doi: 10.21037/cdt.2016.11.19. Review. PubMed PMID: 28123973; PubMed Central PMCID: PMC5220205. Oliveira IS, Hedgire SS, Li W, Ganguli S, Prabhakar AM. Blood pool contrast agents for venous magnetic resonance imaging. Cardiovasc Diagn Ther. 2016 Dec;6(6):508-518. doi: 10.21037/cdt.2016.12.05. Review. PubMed PMID: 28123972; PubMed Central PMCID: PMC5220192. Karande GY, Hedgire SS, Sanchez Y, Baliyan V, Mishra V, Ganguli S, Prabhakar Ther. 2016 Dec;6(6):493-507. doi: 10.21037/cdt.2016.12.06. Review. PubMed PMID: 28123971; PubMed Central PMCID: PMC5220209. Vaduganathan M, Patel NK, Lubitz SA, Neilan TG, Dudzinski DM. A "Malignant" Arrhythmia: Cardiac Metastasis and Ventricular Tachycardia. Tex Heart Inst J. 2016 Dec 1;43(6):558-559. doi: 10.14503/THIJ-15-5189. PubMed PMID: 28100983; PubMed Central PMCID: PMC5179170. Sonis JD, Snchez Y, Benjamin Harvey H, Yun BJ, Raja AS, Prabhakar AM. Communication of imaging recommendations … Continue reading →

Highlights of Cardio 2018 Conference Series LLC Ltd is esteemed to invite you to join the26th International Conference & Exhibition on Cardiology and Cardiovascular Medicinewhich will be held from November 12-13, 2018 Sydney, Australia which includes prompt Keynote Presentations, Oral Talks, Poster Presentations AndExhibitions. Summary:Cardio2018(Conference) Is To Meet Top & Best Cardiologists, Doctors,CardiologyExperts, Cardiac Surgeons, Vascular Medicine Specialists, Physicians, Health Care Professionals AtCardiology, Atherosclerosis, ClinicalCardiologyArrhythmia, Cardiac Surgery,Echocardiography, Cardiovascular Medicine, Arrhythmia, Cardiac Surgery Conferences, Events, Annual Meetings , Program From Clinics, Hospitals From Asia, Americas, Europe, USA, Middle East.Cardio 2018has been designed in an interdisciplinary manner with a multitude of tracks to choose from every segment and provides you with a unique opportunity to meet up with peers from bothindustryandacademiaand establish a scientific network between them. We cordially invite all concerned people to come join us at our event and make it successful by your participation. A comprehensive range of topics will be discussed in the event including maternalClinical Cardiology, Cardiac Pharmacology, Cardiac Regeneration & Remodeling, Heart Stroke,Cardio Metabolic Health,Cardio-Oncology, Cardiovascular Epidemiology,Cardiovascular Nursing, Cardiovascular Radiology And Intervention, Cardiac Imaging & Anesthesia, Critical Care Cardiology,Heart FailureAndCardiomyopathies, Pediatric Cardiology,Cardiac Arrhythmia, Cardiac Diseases, Cardiac Surgery. It is our ideology to bring maximum exposure to … Continue reading →

Distinguished ProfessorDepartment of BioengineeringAdjunct Distinguished ProfessorDepartment of MedicineUniversity of California San Diego9500 Gilman DriveLa Jolla, CA92093-0412 Email: Dr. Andrew McCulloch is Distinguished Professor of Bioengineering and Medicine at the University of California San Diego, where he joined the faculty in 1987. He is member of the UCSD Institute for Engineering in Medicine, the Qualcomm Institute, a Senior Fellow of the San Diego Supercomputer Center, and a member of the UCSD Center for Research on Biological Systems. Dr. McCulloch is a Principal Investigator of the National Biomedical Computation Resource and the Cardiac Atlas project, and Co-Director of the Cardiac Biomedical Science and Engineering Center at UCSD. He served as Vice Chair of the Bioengineering Department from 2002 to 2005 and Chair from 2005 to 2008. Dr. McCulloch is Director of the UCSD Interfaces Graduate Training Program and the Interdisciplinary Ph.D. Specialization in Multi-Scale Biology. Dr. McCulloch was educated at the University of Auckland, New Zealand in Engineering Science and Physiology receiving his Ph.D. in 1986. Dr. McCulloch was an NSF Presidential Young Investigator and is a Fellow of the American Institute for Medical and Biological Engineering and a a Fellow of the Cardiovascular Section of the American Physiological Society. He has … Continue reading →

Cardiac remodeling is a term that refers to changes in the hearts size and shape that occur in response to cardiac disease or cardiac damage. When doctors talk about remodeling, they are usually talking about the left ventricle, though occasionally this term is applied to other cardiac chambers. When you remodel your house, that is often considered a good thing. The opposite is true with remodeling of the heart. In general, the greater the degree of ventricular remodeling, the worse the patients outcome is likely to be. When the left ventricle is damaged -for instance, by amyocardial infarction(heart attack) or by cardiomyopathy-changes often occur in the size and shape of the ventricle. The ventricle tends to become enlarged, its general shape becomes more globular and less elliptical, and the muscular wall of the ventricle often becomes thinner. This remodeling occurs due to mechanical stress on the heart muscle produced by the underlying disease process. In the early stages of a heart attack, some degree of remodeling can help the ventricle compensate for the damage that has occurred. But if this initial remodeling process continues, and the changes in the size and shape of the ventricle become more exaggerated, cardiac function … Continue reading →